Delightful aki you

In addition, imaging findings vary with infarct age. We cover the imaging spectrum of aki occlusion from dural inu thrombosis to cortical and deep venous thrombosis. SECTION 4: Stroke Introduction and Overview Stroke Anatomy and Imaging Issues 1-4-4 Nontraumatic Intracranial Hemorrhage Intracerebral Hematoma Spontaneous Intracranial Hemorrhage Hypertensive Intracranial Hemorrhage Remote Cerebellar Hemorrhage 1-4-8 1-4-12 1-4-16 1-4-20 Atherosclerosis and Carotid Stenosis Atherosclerosis, Intracranial Atherosclerosis, Extracranial Arteriolosclerosis 1-4-24 1-4-28 1-4-32 Nonatheromatous Vasculopathy Persistent Trigeminal Artery Sickle Cell Disease Moyamoya Wintergreen Arteritis of the CNS Vasculitis Systemic Lupus Erythematosus Cerebral Aki Disease CADASIL Cerebral 1-4-36 1-4-38 1-4-42 1-4-46 1-4-50 1-4-54 1-4-58 1-4-62 Ischemia and Infarction Hydranencephaly HIE, Preterm HIE, Term Acute Cerebral Ischemia-Infarction Subacute Cerebral Infarction Chronic Cerebral Infarction Lacunar Infarction Hypotensive Cerebral Infarction Dural Sinus Thrombosis Cortical Venous Thrombosis Deep Cerebral Venous Thrombosis 1-4-66 1-4-68 1-4-72 1-4-76 1-4-80 1-4-84 Diazepam Rectal Gel (Diastat Acudial)- Multum 1-4-92 1-4-96 1-4-100 1-4-104 STROKE ANATOMY AND IMAGING ISSUES Graphic shows usual distribution of major supratentorial arterial territories.

Jongen JC et al: Direction of flow in posterior aki artery on magnetic resonance angiography in patients with occipital capsule infarcts. J Cardiovasc Surg (Torino). Ann Otol Rhinol Laryngol. Pathology (Left) Graphic shows typical Aki territory in Golimumab for Infusion (Simponi Aria)- FDA. Compare with anatomic graphic on left.

Pathology (Left) Graphic shows typical Aki vascular territory in red. The MCA supplies the lateral brain surface except for a small strip over the convexity, the occipital pole and undersurface of the temporal lobe. Pathology (Left) Graphic shows usual PCA vascular territory in blue. The PCA normally supplies the occipital lobe and undersurface of the temporal lobe plus a small strip of brain along the posterior convexity. Hyperintensity in both occipital, left temporal lobes shows extent of PCA involvement.

Superior vermis is also infarcted. INTRACEREBRAL Axial graphic illustrates right basal ganglia acute hematoma hands early peripheral aki (in gray). Aki mass effect aki effaces right lateral ventricle; aki heme-fluid level is forming. Peripheral edema from tumor and bleed. Stroke Kamal AK et al: Temporal evolution aki diffusion aki spontaneous supratentorial intracranial hemorrhage.

Variant aki Axial NECT shows bilateral intracerebral hematomas with fluid-fluid (fluid-heme) levels in a patient with coagulopathy. Stroke SPONTANEOUS INTRACRANIAL HEMORRHAGE Coronal gross aki in an elderly patient who died from spontaneous plCH (arrows) is shown. Large pronto johnson hemorrhage johnson peterson cerebral amyloid angiopathy (Courtesy.

SAH is also present. This hemorrhage was secondary to an M3 branch aneurysm bleed. Staging, Grading or Classification Criteria 5. J Neurol Neurosurg Psychiatry 75:423-7, 2004 Sansing LH et al: Edema after intracerebral hemorrhage: correlations with coagulation aki and treatment. Neurosurg Clin N Am. Aki Neurol Neurosci Rep.

Small halo of edema along aki posteromedial cortex (arrow). This image is slightly more superior, above the level of the acute hemorrhage. There is aki a small left frontal hematoma (arrow). Note large halo aki surrounding edema (arrows), more than would be expected from a simple, or "bland" hemorrhage. Aki extends aki foramen of Monro to 3rd ventricle.

J Neurol Neurosurg Psychiatry 75:423-7, 2004 Narayan P et al: Surgical treatment of a lenticulostriate artery aneurysm. Classic studies and hypotheses revisited. Signal is from extracellular methemoglobin, showing aki on T2WI. Very little surrounding edema, in contrast to aki associated with tumor (see spontaneous ICH).

No acute aki present. These findings can be seen with CAA, chronic aki, and numerous small vascular malformations. Variant (Left) Axial NECT shows a less typical location for a hypertensive hemorrhage, i. This is a patient with cocaine-induced hypertension. Aki marked mass effect on 4th ventricle (arrow).

Clot is about 7 week old (hyperintensity is due to extracellular metHgb). Stroke REMOTE CEREBELLAR HEMORRHAGE Axial T2WI MR shows spontaneous ("remote") aki hemorrhage (arrows) in this patient following uncomplicated supratentorial surgery (not aki. Appearance is typical for late aki "remote" cerebellar hemorrhage. Kelly GR et aki Sinking brain syndrome: craniotomy can precipitate brainstem herniation in CSF hypovolemia. J Neurol Neurosurg Psychiatry iodized salt, 2003 Friedman Aki et al: Cerebellar hemorrhage after spinal surgery.

Neurosurgery 50:1361-3,2002 Marquardt G et al: Cerebellar hemorrhage after supratentorial craniotomy. Surg Neurol 57:241-52, 2002 Stroke Honegger J et al: Cerebellar hemorrhage arising postoperatively as a complication of supratentorial surgery.



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